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For comparison, we treated cells with tunicamycin (TUN, a widely-used ER stress inducer) which did not alter phospho-GCN2 (T899), but reduced the mobility of PERK, indicative of its increased phosphorylation (Fig

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The supplemental intravenous form leads to increases of GSH in the gut, but not in tumors, in a sarcoma-bearing rat model

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