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The presence of bacterial endotoxins such as lipopolysaccharide or anticoagulants in the dialysate triggers formation of oxidative species [5153]

Conversely, impaired CoQ 10 trafficking or compartment-specific depletion of ubiquinol may allow lipid peroxidation to originate in one organelle and propagate to others, potentially through membrane contact sites

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Further, oxidative stress leads to inflammation, and vice versa, in a vicious cycle

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