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An in vitro protocol to study the effect of hyperglycemia on intracellular redox signaling in human retinal pigment epithelial (ARPE-19) cells

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After the subcutaneous administration of GLP-1, it could enter the brain by binding to bloodbrainbarrier-free organs such as the subfornical organ and the area postrema [43], or through the choroid plexus, which has a high density of GLP-1 receptors [17]

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In neoplastic cells, activation of NF-B provides the ability to survive by upregulating anti-apoptotic genes, including several members of the BCL-2 family [192], and increases resistance to chemotherapy by controlling the expression of multidrug resistance gene 1 (mdr1) [193]