Immunohistochemistry of liver sections showed CD36 as located in the hepatocyte plasma membrane in patients with steatosis and NASH, compared to a weak CD36 expression confined to the cytoplasm of hepatocytes in normal livers, suggesting that the translocation of CD36 protein from cytoplasm to membrane may be a triggering event in NAFLD progression [25]
Together, these results demonstrate that KG drives resistance to DNA-damaging agents
Br J Haematol 122(6):10011008 Wambebe C, Khamofu H, Momoh JA et al (2001) Double-blind, placebo-controlled, randomised cross-over clinical trial of NIPRISAN in patients with Sickle Cell Disorder
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It discusses how they integrate into personalized medicine and the role of lab testing to inform use
Prospective analysis of circulating metabolites and breast cancer in EPIC