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On one hand, excessive and sustained M1-type microglial responses exacerbate neuroinflammation and induce neuronal hyperexcitability

PMID: 14999221

Prolonged inhibition of DPP may prevent the formation of GLP-1 (9-36) amide, and might ultimately augment oxidative stress in the heart, vasculature, and liver, and promote hepatic glucose production, which would be undesirable in the treatment of obesity-related diabetes

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Weekly weigh-ins are encouraged for accountability and progress tracking

Better Health Beyond weight loss, GLP-1 therapy supports overall wellness by improving metabolic health, energy levels, and insulin sensitivity