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Throughout, we've cited only peer-reviewed studies, and we're careful to distinguish what's proven in humans from what's still emerging from the lab

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Conclusions This study provides a comprehensive examination of the role of mitochondrial dysfunction in DE, delving into its underlying mechanisms and the therapeutic strategies currently targeting this dysfunction

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The immune response in the CL environment receives precise control through histone modifications which include histone 3 lysine 4 trimethylation (H3K4me3) and histone 3 lysine 27 trimethylation (H3K27me3) (158) that either activate or repress cytokine gene expression (98)

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