These LCACs enhance the ability of RAW264.7 cells to phagocytose lipids by upregulating macrophage receptor with collagenous structure (MARCO), thereby aggravating the accumulation of intracellular lipids, which promotes the formation of foam cells and accelerates the development of atherosclerosis [228]
These applications represent off-label use, and the evidence is primarily from small trials and observational studies
Neuroendocrine Function and Response to Stress in Mice With Complete Disruption of Glucagon-Like Peptide-1 Receptor Signaling
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