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Clinical trial data and FDA labeling information indicate that gastrointestinal effects are dose-dependent and typically most pronounced during initial treatment or dose escalation

Improved insulin sensitivity reduces the inflammatory burden on blood vessels, and reduced triglycerides lower the atherogenic lipid profile

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Conversely, reversal of GLP-1(936)s glucagonostatic effect might explain the dramatic elevation of circulating glucagon observed after pharmacological/genetic inhibition of the GCGRs [43, 53, 65, 66], an effect that has been attributed to AA-induced stimulation of alpha cell proliferation

cdc.gov/cancer/obesity/index.htm

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