This clearly suggested that the radioprotective functions of DSePA were through its incorporation into GPxs via increasing the bioavailable selenium pool rather than acting as a GPx mimic [61, 62, 84]
Parallel in vitro studies showed increased eNOS and expression mediated by AMPK and Phosphoinositide 3-Kinase / Protein Kinase B signaling pathways, implicating a GLP-1R/cAMP/protein kinase A (PKA) axis in the upregulation of endothelial NO production (51)
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Concurrently, cAMP/PKA signaling induces autophagic flux and upregulates antioxidant defense mechanisms, facilitating the clearance of damaged organelles and misfolded proteins
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