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GLP-1 RAs exerted neuroprotective effects by inhibiting glutamate accumulation to prevent excitotoxicity and neural apoptosis [39], which possibly explains the protective effects of the GLP1R gene on background DR and severe nonproliferative DR, but not proliferative DR

As discussed above, lipopolysaccharides (LPS) could prompt inflammation and amyloidogenesis in the brain, by inducing the disorder of TNF-a, IL-1, and cyclooxygenase-2 (COX-2) (87)

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Published in 2002, it generated headlines about breast cancer and heart disease risk from HRT that shaped clinical practice and patient behavior for over 20 years

More research is needed in the perimenopause population to further assess risks and benefits, she says