[DOI] [PubMed] [Google Scholar] Alimpiev S., Grechnikov A., Sunner J., Karavanskii V., Simanovsky Y., Zhabin S., Nikiforov S
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Numerous studies underscore their capacity to downregulate lipogenic transcription factors (such as SREBP-1c and ChREBP), activate lipid oxidation pathways via AMPK and PPAR, enhance mitochondrial function, and reinforce antioxidant defenses through upregulation of Nrf2-mediated gene expression
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This procedure is not expected to evaluate the implication of melatonin or the pineal gland in the mechanism of action of the antidepressants, but rather to produce a proxy of the immediate events that follow the consequences of antidepressants at synaptic level
