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Ansar, H., Mazloom, Z., Kazemi, F

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In young mice, it suppressed the release of pro-inflammatory cytokines from macrophages, indicating a potential role in tempering an overactive immune response during youth [44]

This suggests that the rise in Srd5a1 expression at the time of puberty is likely due to the increase in E2 levels and that the early life adversity either functions independently to repress Srd5a1 or might interfere with this activation

We then evaluated the inhibitory activities of the modified peptides (Figure