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This information may help provide context for understanding why some individuals experience robust GLP-1 response while others show modest effects

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In contrast, another study demonstrated that transgenic mice with a beta-cell specific deletion of the GLP-1R have impaired intraperitoneal (ip) glucose tolerance but maintain normal oral glucose tolerance (Smith et al., 2014)

Although extensive research has identified numerous neurobiological underpinnings of AUD, more recent advances in animals and humans reveal that the GLP-1 pathway plays a role