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The hair growth status is shown in Figure 2A

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J Orthop Translation

These data clearly show that the anorexigenic effects of cabergoline depend on GHSR1a and not on ghrelin, providing more evidence that GHSR1a has a central role even in the absence of the ghrelin peptide

FIGURE 3 In summary, this evidence suggests that the origin of excess ROS production is the result of (1) defective mitochondria (due to the abnormal and reduced distribution of cardiolipins) with high intermembrane electrical potential and (2) reduced mitophagy process (due to high expression and activity of P53 protein)