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La capacidad del GSH para mantener la homeostasis inmunitaria es enorme

Additionally, cancer cells carrying a mutant TP53 protein exhibit increased CHAC1 levels and show greater resistance to ferroptosis (Leu et al., 2019

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As GSH cannot readily cross the cellular membrane due to its biochemical properties 35 , we assume that GSH excretion is negligible and do not account for it in the equation

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Similar content being viewed by others Introduction Site-specific protein phosphorylation is one of the most important post-translational modifications (PTMs) as it can rapidly modulate a proteins function by changing its activity, subcellular localization, interactions, or stability 1