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[5] [6] The GIP receptor component is thought to contribute to metabolic and appetite-related effects, whilst glucagon receptor activation may increase energy expenditure and promote fat breakdown (lipolysis) in the liver and adipose tissue
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Additionally, upregulation of B regulatory cells (Bregs) and IL-10 could serve as another mechanism of fueling potential tumorigenesis (123)
dulaglutide (Trulicity)
