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The excess production of ROS is one of the main molecular causes that contributes to varicocele-related male infertility, in which MIR210HG and MLLT4-AS1 (hypoxia-related lncRNAs) also showed significant positive correlations with ROS and negative correlations with sperm count and motility [48]

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Figure 1 depicts two GLP1R-expressing human control tissues as complementary validation of the GLP1R immunoreactivity, as revealed by the MAb 3F52 (Figure 1a and e), together with GLP1R in situ ligand binding as shown by autoradiography (Figure 1bd and fh)

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