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Combined triad-GSH metabolism model simulations: impact of quinone metabolism on glutathione homeostasis The model was used to investigate how single vs constant 50 M exposure of doxorubicin influenced blood and cytosolic GSH and cysteine (Cys) levels, over a 10-hour time-span, shown in Fig

They're signals, not government endorsements

106 found that patients with acute intrahepatic cholestasis had subnormal serum GGT activity despite high bilirubin levels, suggesting that factors inhibited the release of the enzyme into the blood stream from the liver

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These results indicate that high nitric oxide levels and hyperglycemia stimulated by oxidative stress may cause neuroinflammation, likely via nuclear factor-B signaling [60]