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Key pathophysiologic mechanisms include: Mucosal barrier dysfunction: Impaired epithelial tight junctions allow bacterial translocation, triggering innate immune activation T-helper cell dysregulation: Predominantly Th1 and Th17 pathways drive chronic inflammation via TNF-, IL-12, IL-23 the basis for biologic therapy targets Transmural inflammation: Unlike UC (mucosal only), CD involves all layers: mucosa submucosa muscularis propria serosa Granuloma formation: Non-caseating granulomas are pathognomonic but present in only ~3050% of biopsies Fibrosis and stricture: Chronic inflammation activates myofibroblasts collagen deposition luminal narrowing obstructive symptoms Fistula formation: Transmural ulcers penetrate serosa form sinus tracts connect to adjacent bowel, bladder, vagina, or skin 7
Myalgic encephalomyelitis/chronic fatigue syndrome: first described complication after gam-COVID-vac vaccine
BAC levels have effects on the bodys physical and mental functions
While the amount of water used to reconstitute a drug is usually small, infants have low reserves and hyponatremia (low sodium) or other imbalances could occur if large relative volumes of hypotonic fluid are introduced
ACUTE SPINAL CORD INJURY (E.G., IVDD OR FCE) Use alongside rest and conventional care
