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Its absence from early protocols usually reflects timing decisions rather than dismissal

In vivo and in vitro cellular studies suggest that thiamine deficiency increases the production of reactive oxygen species (ROS), generating oxidative stress in the brain and neuronal tissues [55,56,57]

Third, the breakdown products of lipid peroxidation, often aldehydes, are also toxic to cells [5]

J., Alkhalaf, A., Bravenboer, B., Feskens, E

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