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It is proposed to work by binding GHRH receptors on pituitary somatotroph cells, prompting the pituitary to synthesize and release its own growth hormone in a pulsatile pattern, with the body's somatostatin feedback loop still intact so the effect is self-limiting rather than a constant hormone flood

Link zur Studie (PubMed) Longer lifespan in male mice treated with a weakly estrogenic agonist, an antioxidant, an -glucosidase inhibitor, or a Nrf2-inducer (2016) Strong R et al., Aging Cell

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No significant effects of an additional RGD coating on HA surfaces were detected in a rabbit model for cementless joint prostheses [154]

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