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This occurs because cortisol preferentially stimulates adipocyte differentiation and lipid accumulation in visceral depots while promoting protein catabolism in skeletal muscle

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The salt bridge between R283 6.55 and E124 3.33 exists in both antagonist-bound and active ghrelin receptor structure, however, in contrast to antagonist compound 21, ghrelin or GHRP-6 pushes the side-chain of R283 6.55 to swing away from the receptor helical core, which induces rotation of TM6 that initiates the cascade of conformational changes in receptor activation (Fig

Rigorous Method Verification: We don't assume a method works