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Integrated analysis of core targets and GO/KEGG enrichment results indicated PFAS influences hyperlipidemia and atherosclerosis through PPAR signaling pathway, nuclear receptor activity, bile acid metabolism, inflammatory responses, and oxidative stress

These differences highlight the interplay between genetic background and environmental exposure

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The subcellular distribution of the S114A mutant favored a more pronounced nuclear localization with significantly less fluorescence in the cytosol compared to the full-length transgene (Fig