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Currently, GLP-1R agonists are mainly targeted on the endogenous GLP-1R signaling system, which causes GLP-1R agonists to have many pharmacodynamic characteristics like GLP-1

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Hyperglycemia increases the production of Amyloid Beta-peptide leading to decreased endothelial tight junction

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Treating this outcome as platform validation invites repetition of historical errors and misallocation of development resources

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