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Evidence that the major degradation product of glucose-dependent insulinotropic polypeptide, GIP(342), is a GIP receptor antagonist in vivo

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Yet, alterations in EEC numbers and secretion of gut hormones have also been observed in intestinal inflammation and were implicated in the changes in feeding patterns often accompanying these conditions (146, 147) (Figure 4)

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Insulin binding to brain capillaries is reduced in genetically obese, hyperinsulinemic zucker rats