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It is known that STAT3/SOCS signaling can promote cellular senescence by activating the p53-induced gene expression [10, 150]

The area to be treated was delimited by visually analysis of fat accumulation

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GLP-1 agonists might protect the heart indirectly through their effects on weight, blood sugar, blood pressure, and inflammation

If that mechanism contributes meaningfully, it would explain why the effect on drinking appears alongside the effect on appetite rather than independently of it