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All of these factors are targeted in drug development in order to increase leptin sensitivity in obese individuals (Fig

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This version of IGF-1 acts directly on skeletal muscle and other tissues to stimulate cellular growth and protein synthesis in test models

Beyond defining a mitochondrial mechanism for fuel switching, this work provides a conceptual landscape for future investigation of metabolic adaptation across diverse physiological contexts, including exercise, distinct fasting paradigms, and hormone-driven metabolic regulation, and offers new insight into the determinants of responsiveness to GLP1R agonist-based anti-obesity therapies