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Actin Sequestration and Cytoskeletal Regulation The principal mechanism of TB-500 involves binding to the N-terminus of G-actin monomers through the LKKTET hexapeptide motif, forming a 1:1 non-covalent complex that sequesters G-actin from the polymerization-competent pool

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Primary Research Vectors Visceral Adiposity: Studying the reduction of white adipose tissue (WAT) and the shrinkage of fat cell size without altering caloric intake in animal models

Equal proportions reflect the equal weight of the two mechanisms in the conceptual framework