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Impact of neutrophil extracellular traps on fluid properties, blood flow and complement activation

Furthermore, ferroptosis exacerbates the progression of OA

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Cathepsin G inhibition by serpinb1 and serpinb6 prevents programmed necrosis in neutrophils and monocytes and reduces GSDMD-driven inflammation

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ASCVD was present in 229,619 inpatients (15.87%), including those with coronary artery disease, history of myocardial infarction, peripheral vascular disease or stroke