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The nutrient displacement theory suggests that the displacement of protective anti-inflammatory lipids, such as omega-3 PUFAs (docosahexaenoic acid, EPA), contributes to increased inflammatory signaling via the disinhibition of phospholipase A2, which normally mediates the production of pro-inflammatory mediators (117)

Stress and illness often involve systemic inflammation that damages follicles

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At the same time, lanosterol accumulation enhances membrane fluidity and increases ROS production, thereby boosting phagocytic activity, bacterial clearance, and survival in murine models of endotoxin shock [34]

The expression of different HSPs could enhance the thermotolerance at the HS sites, permitting the plants to sustain cell functioning during times of stress (Kotak et al

Poon, R