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Hyperexcitability of motor nerve terminals can be incited by disrupted energy metabolism, ischemic damage resulting from reduced intravascular volume, and electrolyte imbalances

Its documented interactions at the cellular level have made it a focal point for labs investigating tissue remodeling, antioxidant activity, and gene expression regulation

To achieve this, these enzymes can bind non-substrate ligands (ligandin function) and/or catalyze the conjugation of glutathione onto the targeted molecules, the latter activity being exhibited by GSTs having a serine or a tyrosine as catalytic residues

But it was the Gerson therapy that helped put the procedure on the map

PubMed , Bioscience Reports , vol