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Moreover, highly selective small molecule inhibitors of USP14 (IU1 and derivatives), exhibit the capacity to increase the activity of the 26S 15,19 , and show affinities towards USP14 compatible with their use in a chimera design 15,19,20

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The metabolic adaptations that occur are limited to the enhanced expression of beta-3 adrenergic receptors in adipose tissue, which actually represents a favorable metabolic improvement rather than a dependence-creating change

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