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Adaptation to oxidative stress: quinone-mediated protection of signaling in rat lung epithelial L2 cells

Gln depletion treatment activates FADD and caspase-8-dependent apoptosis involving pro-apoptotic TNF-related apoptosis-inducing ligand receptor 2 (TRAIL-R2) in tumour cells, while metabolic stress induced by glutamine deprivation leads to the downregulation of the GCN2-non-dependent FLICE-inhibitory protein (FLIP), which promotes caspase-8 activation and apoptosis [55]

Clinical studies show benefits at both ends of this range, with higher doses producing more dramatic results

Air pollution and chemical exposure increase toxin load

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