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10.1371/journal.pone.0146797 Summary Keywords autism, folinic Acid, glutathione, methylation, methylcobalamin, oxidative stress, tetrahydrobiopterin Citation Delhey LM, Tippett M, Rose S, Bennuri SC, Slattery JC, Melnyk S, James SJ and Frye RE (2018) Comparison of Treatment for Metabolic Disorders Associated with Autism:Reanalysis of Three Clinical Trials

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In the first shuttle, cytosolic NADH is oxidized to NAD + by MDH1 in conjunction with the reduction of oxaloacetate (OAA) to malate

In this cell line glutamate induced cytotoxicity could be mediated by oxidative stress through CySS/glutamate antiporter, depletion of GSH, down regulation of SOD activity leading to apoptosis (oxidative glutamate toxicity

Further, changes in glucose metabolism regulated ROS production and inflammation, particularly inflammasome-mediated inflammation, in both human and rodent myeloid cells (Zhou et al., 2011