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Cancer cachexia was induced in colon-26 tumor-bearing BALB/c mice, as described in a previous study by our group (3)

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Ammonium metabolic reprogramming in prostate cancer constitutes a pathogenic cornerstone that drives tumor initiation, progression, and therapeutic resistance through its dual role as a metabolic integrator and microenvironmental modulator (103)

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Conversely, when SAM concentrations are low and MTHFR co-factor levels are high, homocysteine remethylation may be increased