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In human CaMKK2, Ca 2+ -calmodulin binding induces autophosphorylation of a threonine residue (Thr85) located in the N-terminal regulatory sequence, which creates a molecular memory that enables CaMKK2 to remain in the activated state following an initial, transient Ca 2+ -signal [33]

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Remarkably, up to 51% of tirzepatide-treated patients reached an A1C below 5.7%, a level reflecting near-normal glycemia

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Norhammar AM, Rydn L, Malmberg K: Admission plasma glucose