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Confirming the activity of the enzyme in mammalian cells, ectopic expression of a codon-optimized GshF cDNA in HEK-293T cells increased cellular GSH levels and conferred resistance to the BSO treatment (Fig

Paracchini V, Chang SS, Santella RM et al (2005) GSMT1 deletion modifies the levels of polycyclic aromatic hydrocarbon-DNA adducts in human sperm

503,512,518 These effects include modulating cell proliferation, inflammation, and oxidative stress in liver cells, all of which are key factors in the development and progression of HCC

Whether the science fully supports every claim is a separate question