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Semaglutide reduces blood glucose through a mechanism where it stimulates insulin secretion and lowers glucagon secretion, both in a glucose-dependent manner

Clinically, long acting GLP-1 mimetics are now used to treat Type 2 diabetes

GLP-1 receptors have been found in the choroid plexus, the very part of the brain that produces cerebrospinal fluid (CSF)

Semaglutide itself does not typically increase bleeding risks, but when taken with aspirin, its essential to monitor for signs of excessive bleeding

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