Although the mechanism(s) underlying NASH progression remain to be fully elucidated, accumulating evidence suggests that both oxidative stress and the pro-inflammatory cytokine tumor necrosis factor- (TNF) are critical contributors in this process 5
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Additionally, AEC-II notably contribute to tissue repair and regeneration by exhibiting the capability to differentiate into AEC-I and promote the regeneration of damaged alveolar structures [35]
Plus, it improved muscle strength and functional power (4)
Injury leads to the appearance of cells with characteristics of both microglia and astrocytes in mouse and human brain
