Taking all this data together it seems logical or appropriate to conclude that, because IGF1-R has a different signalling pathway that can maintain lipid oxidation in the liver, FFA uptake in muscle, and activates mTORC1 could reactivate IR through tyrosine kinase activity on IRS, thus displacing serine phosphorylation, reinstituting insulin signalling
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EHF: extended high frequency
This is why it is important to eat foods with a high carnitine content, such as meat and eggs, in order to maintain an optimal nutritional status
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