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The healing cascade involves several converging mechanisms

The above carbon source-dependent phenotypes and physiological alterations directly or indirectly regulate the pathogenicity of C

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The loading phase (Weeks 14) uses twice-weekly dosing to establish tissue saturation, followed by a maintenance phase (Weeks 512) at once weekly [1]

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These results suggest that PET particles at this concentration do not cause oxidative stress, but rather trigger a temporary mitochondrial dysfunction followed by recovery, likely through mitohormesis [133]