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to reduce (or prevent) the conversion of testosterone to oestrogen (aromatase inhibitors such as anastrozole), to selectively block the action of oestrogen in breast tissue (Selective Estrogen Receptor Modulators (SERMs) such as tamoxifen) and to stimulate endogenous testosterone production following an AAS cycle (tamoxifen, clomiphene, hCG)

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GHK-Cu Levels in various tissues Source: PubMed Research in rats indicates that one possible mechanism by which GHK-Cu protects brain tissue is by preventing apoptosis

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