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These functional protective effects are consistent with metaproteomic findings in children with recent-onset T1D (188), where gut microbiota dysregulation (including reduced butyrate-producing taxa) correlates with -cell damage severity, supporting a clinical link between butyrate deficiency and T1D pathogenesis
L., Green, R
PMID: Summary Keywords acute pancreatitis, inter-organellar interactions, mitochondria, mitochondria-associated membranes, pancreatic acinar cells Citation Chooklin S and Chuklin S (2026) Tangled contacts: mitochondrial interactions with cellular organelles in acute pancreatitis
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Humphries, J
Role of oncometabolites in tumor progression As mentioned above, among the hallmarks of cancers, metabolic alterations are emerging as one of the most intriguing and complex therapeutic targets [21, 22] due to the strict connections of the different metabolic pathways and the capacity of cancer cells to adapt their energetic requirements on nutrient availability and microenvironmental changes
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