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[DOI] [PMC free article] [PubMed] [Google Scholar] 17.Colin IM, Grard KM
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Since these fibrinaloid microclots are usually more resistant to fibrinolysis, they are hypothesised to linger in the circulation and have extended contact with the endothelium (313)
Vitamin B 12 deficiency results in megaloblastic anemia, GI lesions, and neurologic damage that begins with an inability to produce myelin and is followed by gradual degeneration of the axon and nerve head
Recent advances have focused on three strategies: senolytics (eliminating senescent cells, e.g., dasatinib + quercetin), senomorphics (inhibiting the senescence-associated secretory phenotype, e.g., rapamycin), and senoreversion (rejuvenating senescent cells via epigenetic reprogramming)
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