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Disabled macroautophagy, loss of proteostasis, genomic instability, epigenetic alterations, mitochondrial dysfunction, cell senescence, dysregulated neurohormonal signalling and inflammation emerge as common molecular hallmarks of cardiovascular ageing

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Each modification solves a specific biological problem, and together they create a molecule with pharmacokinetic properties no natural peptide possesses

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Symptoms include fatigue, brain fog, and tingling in the extremities all of which overlap with common GLP-1 side effects, making deficiency easy to miss

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