This treatment alleviated lung histological alterations and suppressed inflammatory cell infiltration, demonstrating GHK-Cu's potential as a novel research subject for ALI and acute respiratory distress syndrome (ARDS) by mitigating excessive inflammatory responses
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Induction of ferroptosis by erastin, C dot, or FAC and BSO leads to its dissemination to neighboring cells through an iron and lipid peroxide-dependent mechanism, whereas the GPX4 inhibitor ML162 fails to transmit the ferroptosis signal to adjacent cells
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