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The relative level of Bax was also significantly reduced in the GO@GSH-FA-treated group, further confirming the anti-apoptotic effect (Figure 11E)
Inhibition of mitochondrial (etomoxir) and peroxisomal (thioridazine) fatty acid oxidation revealed that each organelle contributes to approximately half of acetyl-CoA produced from palmitate in the DKO cells (Supplemental Figure 5G)
Always consult your healthcare provider regarding injection technique, site selection, and any concerns about your medication
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Cornelis T, Claes K, Gillard P, Nijs E, Roskams T, Lombaerts R, Nevens F, Cassiman D (2008) Cholestatic liver disease in long-term infantile nephropathic cystinosis
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