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10.1016/j.neuro.2012.10.018 74 GuthrieG

Tumor acidification and GSH depletion by bimetallic composite nanoparticles for enhanced chemodynamic therapy of TNBC

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The investigators reported that LR3 showed the lowest IGFBP-binding affinities of the analogs studied while retaining full IGF-1R binding, supporting the molecular model of the bioavailability mechanism

BPC-157: CAS 137525-51-0, formula CHNO, MW 1,419.55 Da

The mechanism involves upregulation of VEGF expression a key growth factor in new blood vessel formation and activation of ERK1/2 signaling pathways that drive cell proliferation, migration, and angiogenesis