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Alterations in the levels of iron, ferritin and other trace metals in Parkinsons disease and other neurodegenerative diseases affecting the basal ganglia

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Current theories concerning disease pathogenesis include an imbalance between protease and anti-protease activity, induced apoptosis of alveolar cells through deregulation of pathways involved in oxidative stress, chronic inflammation, and aberrant tissue remodeling that lead to the destruction of the extracellular matrix (ECM) in the lung [3, 4]

This suggests that OXPHOS acts as a moderator of hemocyte function under normal conditions