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Retatrutide's mechanism of action involves the stimulation of three crucial hormone receptors: GLP-1 (Glucagon-like peptide-1): Plays a key role in lowering blood sugar levels and promoting satiety, which helps reduce food intake

But theres a second mechanism at play

The signature of class B GPCRs is a ~15 kDa N-terminal extracellular domain (ECD) essential for binding to the C-terminal part of the cognate peptide hormones

The article says semaglutide activates GLP-1 receptors in the pancreas and gastrointestinal tract, slowing gastric emptying and reducing appetite, while statins inhibit HMG-CoA reductase to block cholesterol synthesis in the liver